An In-Depth Investigation of Diverse Therapeutic Benefits of Psidium guajava with Major Emphasis on Anti-Diabetic effect

 

Betsy Sunny1*, Dr. Manju Maria Mathews2, Dhanish Joseph3, Flowerlet Mathew4, Junia George5, Bimi Varghese6

1,5,6Department of Pharmaceutics, Nirmala College of Pharmacy, Muvattupuzha, Kerala

2Associate Professor, Department of Pharmaceutics, Nirmala College of Pharmacy, Muvattupuzha, Kerala 3,4Assistant Professor, Department of Pharmaceutics, Nirmala College of Pharmacy, Muvattupuzha, Kerala *Corresponding Author E-mail: betsysunnye@gmail.com

 

ABSTRACT:

Herbal remedies, or phytotherapy, is the science of using herbal medicines to treat the sick. Natural herbs are the highly esteemed source of medicine throughout human history. Psidium guajava (PG), the common Guava tree popularly known as 'poor man's apple of the tropics is an important dietary plant having a long history of traditional use for a wide range of ailments. The medicinal properties of guava fruit, leaf and other parts of the plant are also well known in the indigenous system of medicine. In view of the immense medicinal importance of PG, this study reveals the efficacy of PG in the amelioration of diabetes, a chronic metabolic disorder that affects the body's ability to produce or use insulin. Treatments could be achieved by the use of synthetic oral hypoglycaemic agents. But due to the major to minor side effects that have been reported so far there is a persistent need to search for safe and effective alternatives.

 

KEYWORDS: Psidium guajava, Diabetes, Chronic metabolic disorder, Oral hypoglycaemic agents, Herbal medicine.

 

 

 

INTRODUCTION:

The practice of using plants as medicines precedes written human history. Herbs for Diabetes treatment are not contemporary. Since ancient times, plants and plant extracts were used to counter Diabetes.1 Diabetes mellitus is a lifetime disease caused by inherited and/or acquired deficiency in the production of insulin by the pancreas, or by the inaction of the insulin produced. Such a deficiency may result in increased levels of glucose in the blood, which in turn impair many of the body's systems, in particular, the blood vessels and nerves. Recently compiled data show that approximately 150 million people have Diabetes mellitus worldwide and that this number may well double by 2025. The management of Diabetes includes the following components of treatment such as Diet and Exercise, Oral hypoglycemic therapy and Insulin therapy.2

 

The Allopathic drugs are often limited in adequacy, carry the risk of unwanted effects, and are often expensive. Whereas herbs represent a vast source of potentially useful dietary supplements for improving blood glucose control and preventing long-term complications in type 2 diabetes mellitus3. Thus the aim of this work is to summarize the studies conducted on both in-vivo as well as in-vitro biological activities of Psidium guajava (PG) by various researchers based on a comprehensive review of pieces literature.

 

METHODOLOGY:

Data from various articles both review as well as research articles were collected from various Databases such as Google Scholar, Pubmed, and Science direct on the different in-vivo and in-vitro studies of PG. Gone through the articles in detail and a better understanding of the various activities with major emphasis on anti-diabetic studies was made.

 

Plant Description:

Guava is a low evergreen tree or shrub of 6 to 25 feet high, with wide-spreading branches and square, downy twigs, is indigenous to tropical America. It is a usual vegetation cover by roads and in waste places in Hawaii. Guava is a tropical and semitropical plant. It is well known in the islands for its edible fruit. It is common in the backyards. The branches are crooked, bringing opposite leaves. The flowers are white, incurved petals, 2 or 3 in the leaf axils, they are fragrant, with four to six petals and yellow anthers. The fruit is small, 3 to 6 cm long, pear-shaped, reddish-yellow when ripe.4

 

Table No.1 Scientific Classification of Psidium guajava5

Taxonomy

Nomenclature

Kingdom

Plantae

Division

Magnoliophyta

Class

Magnoliopsida

Subclass

Rosidae

Order

Myrtales

Family

Myrtaceae

Subfamily

Myrtoideae

Tribe

Myrteae

Genus

Psidium

Species

Psidium guajava

 

Chemical Constituents7:

Guava hold in a wide range of phytochemicals including polysaccharides, vitamins, minerals, essential oils, amino acids, proteins, enzymes, sesquiterpenoid alcohols, triterpenoid acids, aromatic esters, alkaloids, glycosides, flavonoids, tannins, and saponins. It is rich in antioxidants and vitamins, high in lutein, zeaxanthin and, lycopene. It also contains ample amounts of tannins, phenols, triterpenes, flavonoids, essential oils, saponins, carotenoids, lectins, vitamins, fiber, and fatty acids. Fruit comprises of vitamin C and contains appreciable amounts of vitamin A as well. Guava fruits are also a good reservoir of pectin - a dietary fiber. Leaves are a rich source of flavonoids, in particular, quercetin. Much of guava's healing potential is attributed to these flavonoids. And the flavonoids have demonstrated antibacterial activity. Quercetin is thought to contribute to the antidiarrheal effect of guava; it is able to relax the intestinal smooth muscles and inhibit bowel contractions.

Table No.2 Nutritional profile of Psidium guajava fruit6

Nutrient

Amount

Vitamin G4

36-50 mg

Calories

77-86 g

Moisture

2.8-5.5 g

Crude fibre

0.9-1.0 g

Protein

0.1 - 0.5 g

Fat

0.43-0.7 g

Ash

9.5-10 g

Carbohydrates

9.1-17 mg

Calcium

17.8-30 mg

Phosphorus

0.30-0.70 mg

Iron

200-400 I.U.

Carotene (vitamin A)

0.046 mg

Thiamine

0.03-0.04 mg

Riboflavin

 0.6-1.068 mg

Niacin

40 I.U.

Vitamin B3

35 I.U.

 

 

Anti-Diabetic Activity of Psidium guajava:

PG has traditionally been used for the treatment of diabetes. The antihyperglycemic activity of the various parts of PG carried out in-vivo are being discussed in the table below.

 

Three types of leaf extracts are being mentioned in the table above. The solvents used for extraction is different in three of the cases. All the extracts were tested on male Wistar rats of almost the same body weight and are compared with the activity of the standard.

 

Improved levels of plasma Insulin and Haemoglobin by leaf extract of Psidium guajava:

In the study conducted on antidiabetic and antioxidant potential of PG leaves, in streptozotocin-induced diabetes in rats mentioned in the oral administration of PG leaf extract (300 mg/kg body weight/day) for 30 days to streptozotocin-induced diabetes rats significantly decreased the levels of blood glucose, glycosylated haemoglobin and improved the levels of plasma insulin and haemoglobin (Table No.3).

 

 

 

Table No. 3 In -Vivo Anti Diabetic Activity of Leaf Extracts of Psidium guajava

Plant

part

Extraction method

Treatment

Animal/ induction method

Dose

In-vivo response

Reference

Glucose level

% Reduction

Leaf extract

500 mL of petroleum ether 60-80°C, 500 ml 95% ethanol

30 Days

Male Wistar albino rats, wt: 160-180 g, STZ: 50 mg/kg, 10% glucose, 6 animals

300 mg/kg

98.31 ± 7.07

68.04

S. Subramanian et al., 3

Gliclazide: 5 mg/kg

107.62 ± 7.2

65.05

Diabetic control

307.62 ± 24.9

-

Control

91.84 ± 5.42

-

Leaf extract

Distilled water, dried at 30° C

30 Days

Adult male Wistar albino rats, wt: 120-150 g, STZ: 60 mg/ kg, 6 animals

500 mg/ kg

127.33± 2.027

64.59

Shadia Ali Radwan et al., 8

Glibenclamide : 5mg/kg

156.5± 1.47

56.96

Leaf extract 500 mg/kg+ Glibenclamide 5 mg/kg

112.33± 1.429

68.76

Diabetic control

359.67± 2.17

-

Control

110 ± 1.39

-

Leaf extract

98% ethanol

9 Hour

Male Wistar rats, wt : 180 ±10 g , alloxan :(100 mg/kg i.p.), 5 rats, 3 group

750 mg/kg

7.22±0.53

75.12

Santosh Mazumdar

et al., 9

Glibenclamide : 4.15 mg/kg

5.36 ± 0.59

81.53

Diabetic control

29.03±1.81

-

Control

6.14±0.22

-

STZ: Streptozotocin

 

Even though the variation is less the leaf extract has shown a more reduction in the blood glucose level as compared to the Standard drug; Gliclazide. The % reduction of the blood glucose level of leaf extract has shown an increase of three times compared to the standard. When compared with the blood glucose level of the Diabetic control a significant reduction in the blood glucose level can be noticed in the case of the petroleum ether and ethanolic leaf extract of PG. The levels of protein, urea, creatinine, non-enzymatic antioxidants, and the activities of enzymatic antioxidants such as superoxide dismutase, catalase, glutathione peroxidase, and glutathione S-transferase were also markedly altered in the liver of STZ-induced diabetic rats. Oral administration of PG for 30 days restored all these biochemical parameters to near control levels.

 

Improvement of Liver and Kidney Functions by leaf extract of Psidium gujava:

Anti-diabetic effect of aqueous extract of guava leaf using rat model of diabetes induced by streptozotocin was investigated by Shadia Ali Radwan et al. (Table No.3). In addition, the effect of this extract on liver and kidney functions induced in the rat model of diabetes were investigated. Rats were divided randomly into control group, rat model of diabetes induced by streptozotocin, rat model of diabetes treated with aqueous extract of guava leaf, rat model of diabetes treated with Glibenclamide and rat model of diabetes treated with aqueous extract of guava leaf plus Glibenclamide. Aqueous leaf extract was given at a dose of 500mg/kg. Standard used here is Glibenclamide 5mg/kg. Diabetes was induced using Streptozotocin 60 mg/kg. The least blood glucose level was shown by the control which is a noticeable fact. The next least blood glucose level was exhibited by the leaf extract and Glibenclamide combination followed by the leaf extract and then the Standard. But coming to the % reduction of glucose Glibenclamide has shown the highest reduction followed by the leaf extract and finally the leaf extract and Glibenclamide combination. Streptozotocin-induced a significant increase in the activities of Aspartate aminotransferase (AST), Alanine aminotransferase  (ALT), Alkaline phosphatase (ALP) and a significant increase in the levels of bilirubin, urea, creatinine and uric acid. In addition, histopathological and immunological changes were detected in the pancreatic tissue. In this rat model of diabetes, a significant decrease in the serum insulin level and a significant increase in glucose level were detected. But the result data revealed that aqueous extract of guava leaf improved the reduced insulin level and the high glucose level induced by streptozotocin. This was associated with an improvement in the changes in the liver and kidney functions. Loss of body weight gain induced by streptozotocin was alleviated by guava leaf extract, glibenclamide or both. The duration of treatment was 30 days.

 

Stimulation of glucose metabolic enzymes in Liver tissues by Psidium guajava leaf extract:

When PG leaf was extracted with 98% ethanol and administered at a dose of 750mg/kg has shown a reduction in the blood glucose level of 75.12% (Table No.3) as leaf extract might have stimulated glucose metabolic enzymes in liver tissues. Standard drug Glibenclamide was given at a dose of 4.15mg/kg. Here Diabetes was induced with Alloxan Monohydrate 100 mg/kg. Though the blood glucose levels were found to have only the least difference the % reduction was found to be higher for the standard.

 

The order of glucose reduction of the leaf extracts as per (Table No.3) is as follows: Ethanolic leaf extract of PG 750mg/kg > petroleum ether and ethanolic leaf extract 300mg/kg > aqueous leaf extract 500 mg/kg.

 

Antidiabetic potential of Lanostene-type triterpenoids of leaves of Psidium guajava:

As leaf extract has proven to have antidiabetic activity the constituents present in the leaf may also possess antidiabetic effect. Thereby a review was made on the study conducted by Priyanka Bagri et al., on Lanostene-type triterpenoids from leaves of PG (Table No.4). The constituents were extracted using 250ml methanol, silica gel, petroleum ether, chloroform, and the study was conducted for 7 days on male albino Wistar rats. Streptozotocin 60mg/kg was used to induce diabetes. Four types of Lanostenoids were tested at the same dose of 50mg/kg and compared with the control. Trihydroxypsidiumlanostenoic acid (50mg/kg/day) has shown the least glucose level of the four Lanostenoids.

 

 

Table No.4 Lanostenoids From Leaves

Plant part

Extraction method

Treatment

Animal/ Induction method

Dose

Glucose

Level

Reference

Lanostenoids from leaves

250 ml methanol, silica gel, petroleum ether, chloroform

7 Days

Male Albino Wistar rats (150-200 g), STZ: 60 mg/kg, 6 rats, 6 group

Psidiumlanostenoic acid (50 mg/kg/day)

100.67 ± 10.71

Priyanka Bagri et al., 10

12β-Hydroxypsidiumlanostenoi c acid (50 mg/kg/day)

87 ± 11.78

Trihydroxypsidiumlanostenoic acid (50 mg/kg/day)

84.50 ± 12.42

Psidiumlanostenoic acid glucoside (50 mg/kg/day)

90.33 ± 12.88

Control

90.34 ± 8.89

Table No.5 Other Parts of Psidium guajava with Anti -Diabetic Effect

Plant part

Extraction method

Treatment

Animal/ induction method

Dose

In-vivo response

Reference

Glucose level

% reduction

Unripe fruit peel extract

Hot water, 48 hr

21 Days

Male albino Wistar rats, 6-8 week old, 150-200 g, STZ: 45 mg/kg wt, 6 rats, 5 group

400 mg/kg

255.7 ± 6.8

21.78

Prashant K. Rai et al., 11

Diabetic control

326.9 ± 6.8

-

Stem bark extract

Ethanol (95%)

3 Hour

Wistar rats (180–200 g) of either sex, alloxan:120 mg/kg, 5 animals, 3 group

250 mg/kg

223.4 ±2.60

36.02

H.M. Mukhtar et al., 12

Gliclazide

308.2 ±2.522

11.74

25 mg/kg

Diabetic control

349.2 ± 2.08

-

 

Table No.6 In-Vitro Antidiabetic Activity of Leaf Extract of Psidium guajava

Plant part

Extraction method

Experimental model

Dose

Movement of glucose out of dialysis tube (mg/dl)

Reference

Leaf extracts

 Ethyl acetate, n-hexane, ethanol, methanol, petroleum ether, and chloroform: Continuous percolation

Dialysis tube: plant extract, CMC, Sodium chloride containing D-glucose, 27 hr incubation period

n-Hexane extract (50g/l)

301.26±1.86

Sabjan Khaleel Basha et al., 13

Ethanol extract (50g/l)

260±1.62

Methanol extract (50 g/l)

201±1.69

Aqueous extract (50 g/l)

212.11±1.44

Control

3142±289

 

 

Psidiumlanostenoic acid glucoside (50mg/kg/day) has shown a result comparable with control. The study concluded that the leaves of PG possessed antidiabetic lanostene-type triterpenoids. The order of reduction of blood glucose levels by the various lanostenoids is: Trihydroxypsidiumlanostenoic acid (50mg/kg/day) >12β-Hydroxypsidiumlanostenoic acid (50mg/kg/day) > Psidiumlanostenoic acid glucoside (50mg/kg/day) >Psidiumlanostenoic acid (50mg/kg/day).

 

Other parts of PG like the fruit as well as stembark also possess antidiabetic property that was proven from the above studies conducted on the unripe fruit peel extract and stembark extract.

 

Glycemic potential of aqueous extract of Psidium guajava unripe fruit peel:

When the unripe fruit peel of PG was extracted using hot water for 48 hrs and given to albino rats at a dose of 400 mg/kg, a reduction in the blood glucose level of 21.78 % was observed (Table No.5). The duration of the study was 21 days. Streptozotocin was induced at a dose of 45 mg/kg. Blood glucose levels were measured by the glucose oxidase method.

 

Enhancement of peripheral metabolism of glucose by Stem bark extract of Psidium guajava:

The stem bark of PG after extraction with ethanol 95% and administered at a dose of 250mg/kg, a 36.02% reduction in the blood glucose was observed (Table No.5). It was compared with Gliclazide 25mg/kg and the significant reduction was shown by stem bark extract 25 mg/kg. Oral administration of the ethanol extract led to significant blood glucose lowering effect in alloxan-induced hyperglycemic rats. The fall was seen at 1 hour and remained up to 3 hours after the administration of the extract whereas the fall in case of Gliclazide administration was marginal. This may be due to the fact that alloxan treatment causes permanent destruction of the β-cells and Gliclazide requires more than 30% functional pancreas for the effect. The hypoglycaemic activity of PG stem bark could be due to an enhancement of peripheral metabolism of glucose.

 

Improvement in Body weight and Lipid profile by Seed extract of Psidium guajava:

A study on the seed extracts of PG done by Kanakam Vijayabhaskar et al.,7 " Evaluation of anti-diabetic activity on guava seeds aqueous extract in streptozotocin-induced diabetic rats" the effect of aqueous and cold extracts of PG seeds, on fasting blood sugar levels and serum biochemical analysis in streptozotocin-induced diabetic rats was investigated. All the extracts of PG produced a significant antidiabetic activity at dose levels of 1/5th of their lethal doses. These extracts also showed improvement in parameters like body weight and lipid profile as well as regeneration of β-cells of the pancreas.

 

Inhibition of Glucose Diffusion by various leaf extracts of Psidium guajava:

In the in-vitro study to evaluate the glucose uptake of crude n-hexane, ethanol, methanol and aqueous leaf extracts of PG all the extracts were subjected to the inhibitory effect of glucose utilization using specific standard in vitro procedure and were tested at the same doses of 50g/l (Table No.6). Duration of the study was 27 hours. Of all the extracts being tested the methanolic extract was found to be more potent than other extracts with the lowest mean glucose concentration of 201±1.69 mg/dl.

 

Other Pharmacological Actions of Psidium guajava:

Antioxidant Activity:

A comparison on the antioxidant activity and free radical scavenging effects of extracts of guava leaves and dried guava fruit were done by Hui-Yin Chen and Gow-Chin Yen S. The inhibition of linoleic acid oxidation was 94.4-96.2% in a concentration of 100 mg/ml. The results prove that the scavenging effect of ABTS and superoxide anion is increased with the increasing concentration of guava leaf extracts. The antioxidant activity may be due to the blocking of chain reaction by the linoleic acid.14

 

Cardioprotective Effect:

Studies conducted by S Yamashiro et al., showed that the aqueous leaf extract of PG exhibited cardioprotective effects against myocardial ischemia-reperfusion injury in isolated rat heart. Elevation of endogenous antioxidants, retention of the myocardial antioxidant status and appreciable restoration of most of the varied hemodynamic parameters may have contributed to its cardioprotective effect.15 Another investigation by Gondim Antonio showed that aqueous fractions collected from the acetic extract of PG leaf exhibit negative inotropic effect by declining the atrial contractility by reduced calcium (Ca2+) entry in myocardial cells and also by opening potassium channels of cardiac tissue.16

 

Anti Inflammatory Activity:

A decoction of PG leaves was used for the treatment of various inflammatory ailments including rheumatism. Anti-inflammatory and analgesic effects of the leaf extracts of PG was due to the presence of polyphenolic compounds and triterpenoids. Aqueous extract of PG at a dose of 50-800mg/kg, i.p. produced dose-dependent and significant inhibition of fresh egg albumin-induced acute inflammation (edema) in rats (JA Ojewole).17 Further, leaf extract (50–800mg/kg, i.p.) also produced dose-dependent and remarkable analgesic effects against thermally and chemically elicited nociceptive pain in mice.

 

Anticancer Effect:

Sang-Bong Lee et al., carried out a study on the anticancer activity of guava branch extracts against HT-29 human colon cancer cells. The study established a program to screen natural products for cytotoxic activity using the MTT reduction assay system to test HT-29 human colon cancer cells. During the course of screening, they found that the acetone extracts of guava branch (GBA) had cytotoxic effects on HT-29 cells. The GBA showed highly cytotoxic effects through MTT reduction assay, LDH release assay, and colony formation assay. In particular, the GBA of the 250µg/ml showed 35.5% inhibition against the growth of HT-29 cells. As expected, GBA induced characteristic apoptotic effects in HT-29 cells, including chromatin condensation and sharking that occurred 24 hours after the cells had been treated at a concentration level of 250µg/ml. To examine the functions on apoptosis, they used a flow cytometric analysis. The apoptotic cells were distributed according to the cell cycle phase shown by sub-G1 DNA content.18

Antitussive Activity:

According to Jaiarj P et al., the water infusion from PG leaves decreased the frequency of coughing induced by capsaicin aerosol as compared to the control, within10 minutes after injection of the extract. These results suggest that guava leaf extract could be recommended as a cough remedy.19

 

Hepatoprotective Activity:

The aqueous leaf extract of PG has been reported for their hepatoprotective action at a dose of 500mg kg-1 by CK Roy et al.,20 Studies conducted by J Gao et al., indicated that the Asiatic acid which is present in the leaves and fruit of PG is responsible for the hepatoprotection.21 According to G Tajua et al., PG leaf extracts (500mg/kg, PO) significantly reduced the elevated serum levels of aspartate aminotransferase, alanine aminotransferase (AAT), alkaline phosphatase, protein and bilirubin, some antioxidant enzymes, Reduced glutathione (GSH), GPx, SOD and CAT activities, were also evaluated in the rats liver homogenate. The higher dose of the extract (500mg/kg, p.o) prevented and showed an increase in liver weight while the lower dose was ineffective in the paracetamol-induced liver damage. In the acute liver injury induced by paracetamol the higher dose (500mg/kg, p.o.) of PG leaf extract was found to be more effective than the lower dose (250mg/kg, p.o.).22 Studies by Prashant Rai Kumar et al., showed that the unripe fruit feel of PG has a potent hepatoprotective agent.23,27

 

Miscellaneous Activity:

Leaf extract of PG is reported in the Acne vulgaris, a chronic inflammatory disease involving colonization of Propionibacterium acnes, plus activation of neutrophils and lymphocytes by F Qadan et al..24 PG leaf extracts have potent antimicrobial activities against Propionibacterium acnes and may be beneficial in treating acne especially when they are known to have anti-inflammatory activities. Also, the aqueous leaf extract of PG has been reported (FA Razak et al., ) to be effective against dental caries and helpful in reducing dental plaque caused by Staphylococcus sanguinis, Staphylococcus mitis, and Actinomyces sp. at a dose of 1mg/ml.25 Guava stem bark and leaf stem extract has been found to possess antigiardiasis activity and inhibit the growth of Entamoebahistolytica by MM Ponce et al., respectively.26

 

 

A-Petroleum ether &ethanolic leaf extract 300 mg/kg, B- Aqueous Leaf extract 500 mg/kg, C-Ethanolic leaf extract 750 mg/kg , D- Aqueous unripe fruit peel extract 400mg/kg, E- Ethanolic stem bark extract 250 mg/kg, F- Leaf extract 500mg/kg +Glibenclamide 5 mg/kg, G- Gliclazide 5 mg/kg , H- Glibenclamide - 5mg/kg, I-Glibenclamide 4.15 mg/kg, J-Gliclazide 25 mg/kg .

Figure 1. % Glucose reduction of different parts of Psidium guajava

 

 

RESULTS AND DISCUSSION:

Based on the data above every part of PG such as the leaf, fruit, bark, and seed possess antidiabetic activity. The studies conducted on the leaf extracts was comparatively more than those of the other parts which undoubtedly provides an idea that leaves are having the best antidiabetic activity than other parts. Leaves were extracted using different solvents such as Petroleum ether, ethanol, water etc. All the parts were used in the form of extracts.

 

From the results obtained a % Glucose reduction VS Dose graph can be plotted. The parts, solvents used for extraction and the doses are given on the X-axis whereas the % Glucose reduction on the Y-axis.

 

A to E are the various plant part extracts and F to J are the different Standard drugs used.

 

From the graph, it is clear that the most potent activity had been shown by (I) Glibenclamide 4.15mg/kg, the standard. 4.15 mg/kg is the least dose of Glibenclamide mentioned in the studies and the second potent activity was shown by the Ethanolic leaf extract 750mg/kg. A combination of the leaf extract and Glibenclamide are also plotted in the graph (F). Of the various parts of PG, leaf extract was found to have the best anti-diabetic potential. From the leaf extracts, ethanolic leaf extract (750mg/kg) was found to be more potent. The potent antihyperglycemic effect of guava leaf extract could be due to its ability to promote glucose uptake by liver and other peripheral tissues together with the inhibition of intestinal glucose absorption induced by guava leaf extract.8 Aqueous unripe fruit peel extract has shown the least glucose reduction amongst the other parts. Of the different standards mentioned to have used in the studies, Glibenclamide 4.15mg/kg was found to be the best. So it can be concluded from the graph that in the case of standard drug, the least dose provides the most potent activity whereas opposite is the case of the extract that is, the highest dose among the extracts showed the most potent activity. Therefore in the case of standard, a low dose is sufficient to provide a marked reduction in the blood glucose level, but for extracts an increase in the dose resulted in a simultaneous increase in activity. The very less activity was shown by Gliclazide 25 mg/kg.

 

In addition to antidiabetic activity, many pharmacological studies have demonstrated the ability of this plant to exhibit antioxidant, hepatoprotective, antimicrobial, cytotoxic, antispasmodic, cardioactive, anticough, and anti-inflammatory activities, supporting its traditional uses.

 

Diabetes is a long-term disease with variable clinical manifestations and progression, chronic hyperglycemia from whatever cause leads to a number of complications including cardiovascular such as hypertension, renal, neurological such as anxiety, stress, ocular and other such inter-current infections. As diabetes is characterized by defects in both metabolic and vascular domains, this disease represents a privileged situation for oxidative stress exerting harmful effects.28 Diabetes adds to the many environmental causes, which are common to many diseases, some extremely active sources generating reactive oxygen species (ROS) as a consequence of high glucose but also, as will be seen, and several other factors.29 Under diabetic conditions, induced oxidative stress also activates the JNK pathway, which is involved in the deterioration of pancreatic β-cell function found in diabetes. There are several novel approaches which might cure diabetes. Defective glucose-stimulated insulin secretion by pancreatic islet β cells could be cured with recombinant glucagon-like peptide 1 (GLP-1) or agonists of the GLP-1 receptor. The role of peroxisome proliferator-activated receptors (PPAR γ) in the regulation of lipid metabolism, insulin and triglycerides lead to the rational design of several PPAR agonists. Gene therapy also generates greater hope for a possible cure for diabetes. Sodium-Glucose Co-Transporter inhibitor is also one of the novel targets for lowering plasma glucose and improving insulin resistance by increasing renal glucose excretion. Treatment with antioxidants and/or suppression of the JNK pathway protect β-cells from some of the toxic effects of hyperglycemia could be the one of novel target therapy of diabetes mellitus.30 Nanotechnology could be made use of in the treatment of diabetes. The stomach acid destroys protein-based Insulin. Diabetic patients control their blood-sugar levels via insulin introduced directly into the bloodstream by injections which is very painful. The new system is based on inhaling the insulin and on a controlled release of insulin directly into the bloodstream. Such kind of treatment for diabetes includes the proper delivery of insulin in the bloodstream it can be achieved by nanotechnology by the development of oral insulin.31 The thiazolidinediones used in oral combination therapy in the management of patients with type II diabetes who have insufficient glycaemic control despite a maximally tolerated dose of oral mono-therapy with either metformin or sulphonylurea.28 Diet therapy is the cornerstone for the management of gestational diabetes mellitus. Although there is widespread use of herbal dietary supplements that are believed to benefit type 2 diabetes mellitus, few have been proven to do so in properly designed randomized trials.32

 

Herbal remedies could provide improved adherence to patients than conventional oral hypoglycemic agents. The quality of herbal drugs is the sum of all factors which contribute directly or indirectly to the safety, effectiveness, and acceptability of the product. A system to ensure that every packet of medicine that is being sold has the correct substances in the correct amount and will induce its therapeutic effect this is known as standardization.33 The major hindrance in the amalgamation of herbal medicines into modern medical practices is the lack of scientific and clinical data and a better understanding of efficacy and safety of the herbal products. It requires a thorough search for medicinal plants, proper guidelines for their identification, validation of the scientific methods of isolation of active ingredients, pre-clinical evaluation of their pharmacological and toxicological profiles, and lastly, the clinical evidence of their usefulness needs to be obtained.34 Other than PG there are numerous other herbs which possess significant hypoglycemic effect. Some of which includes; the methanolic extract of different parts of Coccinia grandis,35 the ethanolic fraction of Thuja occidentalis (EFTO) which exhibited significant anti-hyperglycemic activities in alloxan-induced diabetic rats.36 This extract has shown improvement in parameters like body weight and lipid profile by enhancing the effect on cellular antioxidant defenses to protect against oxidative damage. Alcoholic extracts of Sida cordifolia had more significantly reduced the blood glucose level after a single dose and nearly equal to standard glibenclamide after prolonged treatment. Al the extracts were given orally at a dose of 250mg/Kg b.w.37 Nelumbo nucifera rhizome and flower extract was found to be a promising antidiabetic agent in STZ induced rats.38

 

CONCLUSION:

It is evident from the Literature survey that the extracts from different parts of PG possess excellent antihyperglycemic status. Of the several extracts, leaf extract was found to be more potent. The presence of tannins, triterpenes, and flavonoids in PG leaves may be partially responsible for its antidiabetogenic properties. Moreover, guava is traditionally being used for the treatment of several other ailments including Diarrhoea, Cough, Hypertension, Sore throat, etc. It is also a digestive and carminative, gives vigor and strength to the heart, lungs, and whole body. Toxicity studies in mice and other animal models, as well as in controlled human studies, showed that both leaf and fruit are safe without any adverse effects. Hence supplementing a balanced diet like Guava health tea may provide health-promoting effects. "A Guava fruit a day keeps a Doctor away"; therefore the future prospects should be aimed at investigating the biodiversity of guava and/or the purification of the different compounds present in guava in order to obtain functional ingredients for additional uses as alternate agents in natural therapeutic approaches.

 

REFERENCE:

1.        Kumari MS et al. Natural herbs vs allopathic drugs: To treat diabetes. Indo American journal of pharmaceutical sciences. 2016; 3(5):415-22.

2.        Diabetes Mellitus. Available from: URL: https://www.who.int/mediacentre/factsheets/fs138/en/

3.        Subramanian S et al. Biochemical evaluation of antihyperglycemic and antioxidant nature of Psidiumguajava leaves extract in streptozotocin-induced experimental diabetes in rats. Pharmaceutical biology. 2009; 47(4):298-303.

4.        Anthony C. "A review of Guava (Psidiumguajava)." Available from: URL: http://dweckdata.com/Published_papers/Psidium_guajava.pdf.

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Received on 04.03.2019           Modified on 10.04.2019

Accepted on 16.05.2019         © RJPT All right reserved

Research J. Pharm. and Tech. 2019; 12(10): 5029-5036.

DOI: 10.5958/0974-360X.2019.00873.4